• Introduction
• Etiology
• Dietary sources of iodine
• Iodine deficiency in animal models
• • IDD in the human life cycle
  • • Iodine deficiency in pregnancy
    • • Brain development in humans
      • Severe iodine deficiency in pregnancy: cretinism and increased fetal and perinatal mortality
      • Mild-to-moderate deficiency in pregnancy
    • Iodine deficiency in the neonate
    • Iodine deficiency in the child
    • Iodine deficiency in the adult
  • SPECIFIC IODINE DEFICIENCY DISORDERS
  • • Endemic goiter
    • • Epidemiology
      • Etiology
      • • Iodine Deficiency
        • Goitrogenic factors
      • Pathology
      • Diagnosis
      • Pathophysiology
    • Endemic cretinism
    • • Epidemiology
      • Neurological Cretinism
      • Pathophysiology of neurological cretinism
      • Myxedematous Cretinism
      • Aetiology and Pathophysiologyof myxedematous cretinism
      • Assessment of iodine status in populations
      • Thyroid size
      • Urinary iodine concentration
      • Thyroid stimulating hormone
      • Thyroglobulin
      • Thyroid hormone concentrations
    • Iodine fortification and supplementation
    • • Iodized salt
      • Iodized oil
      • Iodized bread
      • Iodized water
    • Current global status of IDD control programs
    • Monitoring the impact ofprograms of salt iodization
    • The risks of excess iodine intake
    • • Iodide goiter and iodine-induced hypothyroidism
      • Iodine-induced hyperthyroidism, IIH
      • Iodine-induced thyroiditis
      • Recent studies in China and Denmark
      • Thyroid cancer